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  1. Dec 10, 2021 · Cortisol is a glucocorticoid hormone that your adrenal glands produce and release. Hormones are chemicals that coordinate different functions in your body by carrying messages through your blood to your organs, skin, muscles and other tissues. These signals tell your body what to do and when to do it. Glucocorticoids are a type of steroid hormone.

  2. Glukokortikoider, även kallade glukokortikosteroider, är en grupp av steroidhormoner som bildas i binjurens bark, varav kortisol är den viktigaste i människor. I andra däggdjur som möss är kortikosteron den viktigaste glukokortikoiden. Glukokortikoiden har en rad viktiga effekter på kroppen varav många är relaterade till stress och ...

  3. Adult Hippocampus. The adult hippocampus is highly enriched in type I ( mineralocorticoid, MR) and type II ( glucocorticoid, GR) glucocorticoid receptors. Despite receptor name, cortisol has ten times greater affinity for MRs than GRs. At basal GC levels, most MRs are activate.

  4. Adrenal insufficiency is a condition in which the adrenal glands do not produce adequate amounts of steroid hormones. The adrenal glands—also referred to as the adrenal cortex—normally secrete glucocorticoids (primarily cortisol ), mineralocorticoids (primarily aldosterone ), and androgens.

  5. Chemical class. Steroids. Legal status. In Wikidata. Mineralocorticoids are a class of corticosteroids, which in turn are a class of steroid hormones. Mineralocorticoids are produced in the adrenal cortex and influence salt and water balances ( electrolyte balance and fluid balance ). The primary mineralocorticoid is aldosterone .

  6. Pages in category "Glucocorticoids" The following 195 pages are in this category, out of 195 total. This list may not reflect recent changes. A. Acrocinonide;

  7. Glucocorticoid deficiency can be caused by inherited genetic disorders that affect the production of cortisol in the adrenal glands, such as familial glucocorticoid deficiency (FGD). [3] FGD is a group of monogenic recessive disorders caused by disease-causing variants in genes involved in cortisol biosynthesis. [4]

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