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3 days ago · A longstanding mystery about the strain of Vibrio cholerae (V. cholerae) responsible for the seventh global cholera pandemic is how this lineage has managed to out-compete other pathogenic variants. The UT team identified a unique quirk of the immune system that protects the bacteria from a key driver of bacterial evolution.
5 days ago · Strain-specific pili enable Vibrio cholerae bacteria to adhere to each other and form aggregates in liquid culture. A new study focuses on strains with less specific, promiscuous pili and suggests a role for contact-dependent bacterial killing in shaping the composition of these aggregates.
4 days ago · Multidrug-resistant pathogenic vibrios are a crisis of concern as they cause multiple illnesses, including gastroenteritis in humans and acute hepatopancreatic necrosis in aquaculture. In the current study, we investigated the prevalence of the beta-lactamase gene CTX-M-group 1 in Vibrio spp. (Vibrio cholerae and Vibrio parahaemolyticus) from the water and sediment of urban tropical mangrove ...
3 days ago · V. cholerae is the model organism for different species of Vibrio. We have previously shown that V. cholerae’s response to sub-MIC antibiotic stress is transposable to other Gram-negative pathogens (78,82), while there are differences between E. coli and V. cholerae, in the response to sub-MIC antibiotics and oxidative stress phenotypes (78 ...
3 days ago · AMPs play an important role in modulating such microbial interactions and can displace competitor strains, facilitating cooperation. 20 For instance, pathogens such as Shigella spp., 21 Staphylococcus spp., 22 Vibrio cholerae, 23 and Listeria spp. 24, 25 produce AMPs that eliminate competitors (sometimes from the same species), allowing them to ...
2 days ago · Various bacterial pathogens that are commonly isolated from patients with diarrhea include E. coli, V. cholerae, Salmonella spp. and Shigella spp as the common ones considered the core diarrheagenic bacteria (Alhaji et al. Citation 2022).
1 day ago · A recent structural analysis of PG activation across various bacteria, including S. pneumoniae, P. aeruginosa, E. coli, V. cholerae, and M. tuberculosis, reveals a relatively conserved “inside-out” mechano-transmission mechanism (Fig. 8). Initially, in the ATP-free resting state, the two NBD domains are spatially separated in the cytosol.
