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Feb 13, 2017 · Entamoeba histolytica occurs in the following forms, the trophozoites, precyst, cyst, metacyst, and metacystic trophozoite. The trophozoite is about 10–60 μm in size with a clear finely granular cytoplasm and a spherical nucleus that shows aggregation of chromatin beads at the nuclear membrane and a centrally located karyosome.
- Nadia A. El-Dib
- naeldib@hotmail.com
- 2017
After the development of DNA analysis of organisms, pathologic amoebae were properly identified in pathological samples and the prevalence of E. histolytica is expected to be less than before.
- Nadia A. El-Dib
- 2017
Entamoeba histolytica is an anaerobic parasitic amoebozoan, part of the genus Entamoeba. Predominantly infecting humans and other primates causing amoebiasis , E. histolytica is estimated to infect about 35-50 million people worldwide. [1]
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E. histolytica has 2 main stages during its life cycle: the trophozoite and the cyst stage. The infectious agent is the cyst and is transmitted through the oral–fecal route via contaminated food or water. Animal models are not available for this stage interconversion, which is essential for disease propagation and pathogenesis. Excystation
- Carolina Mendoza Cavazos, Laura J. Knoll
- 2020
Aug 19, 2019 · A large number of transcriptome-level studies in Entamoeba histolytica, the protozoan parasite that causes amoebiasis, have investigated gene expression patterns to help understand the pathology and biology of the organism.
Jun 3, 2020 · The life cycle of Entamoeba histolytica is relatively simple, consisting of two stages: the dormant cyst and the vegetative trophozoite ( FIGURE 1 ). Transmission of amoebiasis begins with the ingestion of mature quadrinucleated E. histolytica cysts found in fecal-contaminated food or water ( 40, 133 ).
Jun 1, 2001 · Recent advances in the development of in vitro and in vivo models of disease, new genetic approaches, the identification of key E. histolytica virulence factors, and the recognition of crucial elements of the host response to infection have led to significant insights into the pathogenesis of amebic infection.
