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- Infection with Borrelia burgdorferi often triggers pathophysiologic perturbations that are further augmented by the inflammatory responses of the host, resulting in the severe clinical conditions of Lyme disease.
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The role of this bacterium, named Borrelia (B.) burgdorferi, as the causative agent of Lyme disease was quickly established as the bacterium was recovered from patients as well as from reservoir hosts, such as the white-footed mouse. Although first recognized because of its association with arthritis, it became clear that arthritis was a late ...
- Figure - PMC - National Center for Biotechnology Information
Schematic representation of the infection process (in black...
- Borrelia burgdorferi, the Causative Agent of Lyme Disease ...
Borrelia burgdorferi is the causative agent of Lyme disease,...
- The Brilliance of Borrelia: Mechanisms of Host Immune Evasion ...
Lyme disease (LD) has become the most common vector-borne...
- Figure - PMC - National Center for Biotechnology Information
B. burgdorferi is the causative agent of Lyme disease and is why this bacteria is so important and being studied. It is most commonly transmitted from ticks to humans. Humans act as the tick's host for this bacteria.
Jan 9, 2020 · In the United States, Lyme disease is caused by the bacteria Borrelia burgdorferi and Borrelia mayonii, carried primarily by black-legged or deer ticks. Young brown ticks often are no bigger than a poppy seed, which can make them nearly impossible to spot. To contract Lyme disease, an infected deer tick must bite you.
Borrelia bacteria are spread to people through the bite of an infected blacklegged tick. ... Lyme Disease Surveillance and Data. Available Lyme disease data including how many people get Lyme disease, and where. For professionals. Clinical Care of Lyme Disease. Treatment best practices for the most common manifestations of Lyme disease.
Mar 2, 2021 · The causative agent, Borrelia burgdorferi sensu lato, is capable of establishing a persistent infection within the host. This is despite the activation of both the innate and adaptive immune responses.
