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  1. Von Willebrand disease (VWD) is an inherited bleeding disorder. People with VWD have a problem with a protein in their blood called von Willebrand factor (VWF) that helps control bleeding. They do not have enough of the protein or it does not work the way it should. For people with VWD it takes longer for blood to clot and for bleeding to stop.

  2. Aug 6, 2009 · It is relatively inexpensive, carries no risk of transmitting blood-borne viruses, and can be used for home treatment. Desmopressin is usually administered at a dose of 0.3 μg/kg body weight by a subcutaneous or intravenous route or at a fixed dose of 150 to 300 μg by a metered-dose intranasal spray.

  3. Jan 3, 2024 · seafood. nuts. some dried fruits, such as raisins. spinach and peas. white beans, kidney beans, and lentils. In addition, eating vitamin C-rich foods can help the body absorb iron from food. Foods ...

  4. Von Willebrand multimers test, which helps to classify the type of von Willebrand disease. Platelet function tests, which determine how well the platelets work. Genetic testing is recommended if one or both parents have von Willebrand disease since there is a 50% chance the child will inherit the disorder.

  5. Von Willebrand disease testing should be postponed until a patient is off high-dose estrogen therapy. Standard or low dose estrogen treatment would be much less likely to interfere with von Willebrand disease testing, and we routinely screen for von Willebrand disease in patients on standard-dose combined contraceptives in clinical practice.

  6. Feb 25, 2019 · There are four main types of von Willebrand’s disease. Type 1. Type 1 is the most common. About 60–80 percent of people with von Willebrand’s disease have type 1.. Type 1 is characterized by ...

  7. Nov 12, 2019 · Abstract. The management of von Willebrand disease (VWD) is based upon the dual correction of the primary hemostasis defect, due to the inherited deficiency of von Willebrand factor (VWF), and of the secondary defect of factor VIII coagulant activity (FVIII:C), due to the loss of binding and stabilization by VWF of this intrinsic coagulation ...

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