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  1. Apr 19, 2018 · an aversive stimulus that can serve as a negative reinforcer of behavior, in severe cases because it causes pain or damage to the experiencing organism and in lesser cases because it is unpleasant.

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  3. Sep 1, 1996 · The principal meaning of stimulus with which we are concerned is an external or internal physical change that induces afferent input in the nervous system, with or without sensory experience or a behavioral response. A noxious stimulus is actually, or potentially, damaging to tissue and liable to cause pain, but does not invariably do so.

    • Fernando Cervero, Harold Merskey
    • 1996
  4. en.wikipedia.org › wiki › NociceptionNociception - Wikipedia

    In physiology, nociception (/ˌnəʊsɪˈsɛpʃ(ə)n/), also nocioception; from Latin nocere 'to harm/hurt') is the sensory nervous system's process of encoding noxious stimuli.

  5. Mar 9, 2017 · To fulfill these functions, a noxious stimulus might induce a percept which, in turn, induces a behavioral response. Here, we investigated an alternative view in which behavioral responses do not exclusively depend on but themselves shape perception.

    • Elisabeth S. May, Laura Tiemann, Paul Schmidt, Paul Schmidt, Moritz M. Nickel, Nina Wiedemann, Chris...
    • 10.1038/srep44083
    • 2017
    • Sci Rep. 2017; 7: 44083.
  6. A noxious stimulus is actually, or potentially, damaging to tissue and liable to cause pain, but does not invariably do so. Some noxious stimuli, particularly in the viscera, do not cause nociceptive responses.

  7. A noxious stimulus is a stimulus strong enough to threaten the body's integrity (i.e. cause damage to tissue). Noxious stimulation induces peripheral afferents responsible for transducing pain (including A-delta and C- nerve fibers, as well as free nerve endings ) throughout the nervous system of an organism.

  8. May 1, 2023 · Acute noxious stimuli (e.g., heat, cold, mechanical force, or chemical stimulation) trigger nociceptors. Acute pain becomes inflammatory pain when the noxious stimulus persists long enough to allow nociceptive neurons to release their pro-inflammatory markers and sensitize or activate responsive cells in their local environment.

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