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Sep 22, 2019 · Peptic Ulcer Disease: Pathogenesis and clinical findings. H. pylori. (gram negative rod bacteria) Toxin release. Inflammatory response. Inhibition of H+ detection in gastric antrum. H+ over-secretion.
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Peptic ulcers are sores in the inner lining of the stomach or upper small intestine. They form when the digestive juices produced by the stomach erode or eat away the lining of the digestive tract. Peptic ulcers may form in the lining of the stomach (gastric ulcers) or just below the stomach, at the start of the small intestine (duodenal ulcers ...
Overview. Peptic ulcers are sores on the inside of the stomach. Or they may be on the inside of the small intestine (such as a duodenal ulcer). They are most often caused by an infection with Helicobacter pylori ( H. pylori) bacteria or use of non-steroidal anti-inflammatory drugs (NSAIDs).
Critical illness, trauma. Gastric ulceration (see Peptic Ulcer Disease slide) ↓ Gastric mucosal defense. (↓ secretion of prostaglandins and mucus) Acid erodes gastric mucosa. Inflammatory cells (primarily neutrophils) infiltrate site of injury. Acute Gastritis. Acute inflammation of gastric mucosa.
The two most common causes of peptic ulcers are infection with Helicobacter pylori ( H. pylori) bacteria and use of non-steroidal anti-inflammatory drugs (NSAIDs), such as aspirin, ibuprofen, and naproxen. H. pylori and NSAIDs break down the protective mucus layer in the stomach or intestine.
Jun 5, 2023 · Peptic ulcer disease (PUD) is characterized by discontinuation in the inner lining of the gastrointestinal (GI) tract because of gastric acid secretion or pepsin. It extends into the muscularis propria layer of the gastric epithelium. It usually occurs in the stomach and proximal duodenum.
