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2 days ago · Within the intracranial arteries, many pathophysiologic processes can contribute to increased stiffness, including atherosclerosis, and may or may not be accompanied by significant stenosis. 6 ICAD might partly mediate a relationship between hypertension and AD pathology. 97 Using transcranial Doppler, Roher et al. found a strong association ...
1 day ago · Intracerebral haemorrhage (ICH) is a cerebrovascular disease that is characterized by haemorrhage within the brain parenchyma and has a mortality rate that can reach 50% within 30 days 1.There is ...
4 days ago · ICD-10-CM Diagnosis Codes. S06.9X0A - Unspecified intracranial injury without loss of consciousness, initial encounter. The above description is abbreviated. This code description may also have Includes, Excludes, Notes, Guidelines, Examples and other information.
3 days ago · Background Idiopathic intracranial hypertension (IIH) is a cause of chronic headaches that are probably driven by raised intracranial pressure (ICP). Cerebral venous sinus pressure is thought to play a role in the underlying pathology, but its relation with intracranial pressure requires further investigation. We aimed to evaluate the concordance between lumbar puncture opening pressure (LPOP ...
3 days ago · With the advent of modern neuroimaging tools, continued bleeding and haematoma expansion have been demonstrated in one-third of patients with ICH in the first few hours and in a further 10% in the 3–24 h after stroke onset [33,34,35]. Whether hematoma expansion is caused by continuous or recurrent bleeding, or both remain unknown.
2 days ago · Brain acidosis also can also cause neuronal injury via glutamate-induced neuronal apoptosis, wherein alterations in the functions of intracellular proteins eventually induce the death of neurons and glial cells [24,25,26]. Furthermore, acidosis induces the activation of neuronal acid-sensing ion channel 1, which can cause neuronal death [27,28,29].
4 days ago · Historical perspectives on sudden deaths in young athletes with evolution over 35 years. Am J Cardiol . 2015;116:1461–1468. doi: 10.1016/j.amjcard.2015.07.072 Crossref
