Nov 13, 2017 · November 13, 2017 - by Alison. Dengue disasters: why storms and earthquakes boost mosquito populations and risks. Hurricanes, earthquakes, tsunamis, typhoons and other natural disasters continue to claim their victims long after they strike.
Apr 24, 2018 · Dengue virus (DENV), an arbovirus transmitted by mosquitoes, has become a major threat to American human life, reaching approximately 23 million cases from 1980 to 2017. Brazil is among the countries most affected by this terrible viral disease, with 13.6 million cases. DENV has four different serotypes, DENV1-4, which show a broad clinical spectrum. Dengue creates a staggering epidemiological ...
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The types of mosquitoes that can spread viruses may increase 2 weeks to 2 months after a hurricane, especially in areas that did not flood but received more rainfall than usual.
Dengue virus (DENV) is the cause of dengue fever.It is a mosquito-borne, single positive-stranded RNA virus of the family Flaviviridae; genus Flavivirus. Five serotypes of the virus have been found, all of which can cause the full spectrum of disease.
Scientists from the Institut Pasteur in Paris and the Institut Pasteur in Cambodia, in collaboration with teams from the CNRS and Inria, have demonstrated that asymptomatic dengue infection in children is associated with activation of the immune system via control mechanisms, resulting in the elimination of viral infection without excessive immune activation.
Apr 18, 2017 · Dengue virus NS2B protein targets cGAS for degradation and prevents mitochondrial DNA sensing during infection. Nature Microbiology , 2017; 2: 17037 DOI: 10.1038/nmicrobiol.2017.37 Cite This Page :
Nov 02, 2017 · 4 tus algo i has Dengue for the 2nd time with simultáneos pericarditis an pleuresy. I esa in tve peruvian amazon and urgently brought the WI med center. Recovered 100% Sep 01, 2017. Identical ...
Dengue fever is a mosquito-borne tropical disease caused by the dengue virus. Symptoms typically begin three to fourteen days after infection. These may include a high fever, headache, vomiting, muscle and joint pains, and a characteristic skin rash.
Dengue fever (DF) is a vectorborne disease caused by 4 closely related dengue viruses (DENV 1–4) [1, 2]. DF is distributed in most tropical and subtropical areas, where Aedes aegypti and/or A. albopictus are abundant . Infection with DENV can also cause dengue hemorrhagic fever (DHF), a syndrome characterized by increased vascular permeability, plasma leakage, hypovolemia, and shock [4, 5]. Although the pathogenesis of DHF is not fully understood, several risks have been reported: secondary infection with heterologous strains [6, 7], primary infection in infants born to dengueimmune mothers , differing virulence of the strain , and differing human susceptibility according to race or genetic factors [10, 11]. Because DENV infections are often clinically inapparent, it is difficult to clarify the epidemiology of DF and dengue pathophysiology without virological support [12, 13]. The natural history of DF involves the time from infection to onset and recovery from disease. How...
Brief historical background. Both dengue studies were performed in the Philippines and involved large numbers of human volunteers recruited from US Army personnel. The first study, conducted by Joseph Franklin Siler, MiltonWeston Hall, and Arthur Parker Hitchens took place in 1924–1925, whereas the latter was organized by James Stevens Simmons, Joseph Harold St. John, and Francois Hiie Kari Reynolds in 1929–1930 (the latter study is available at: http://plaza.umin.ac.ip/infepi/simmons1.pdf). Both studies were originally published in the Philippine Journal of Science [23, 24] and were reprinted with appendices by the Bureau of Printing, Manila [25, 26]. In 1971–1972, Halstead obtained blood samples from 4 and 5 volunteers involved in each study and demonstrated specific neutralizing antibodies to DENV-4 and DENV-1, respectively . Following these experimental studies of dengue, J. F. Siler (1875–1960) reported the efficacy of antityphoid vaccines , whereas J. S. Simmons (1890–...
Descriptive characteristics. Among those who developed clinically apparent DF, mean ± SD ages were 22.5 ± 4.0 and 22.0 ± 3.4 years in the DENV-4 and DENV-1 studies, respectively. Age was not significantly different between studies (P = .97). The mean ± SD durations of fever were 3.6 ± 1.2 and 4.8 ± 1.2 days, respectively. The febrile period of DENV-1 infection was significantly longer than that of DENV-4 (P < .01). In the DENV-1 study, the mean ± SD values for minimum WBC count and duration of leukopenia were 3.01 × 109 ± 0.78 × 109cells/L and 6.1 ± 2.0 days, respectively, and 37.5% (95% CI, 27.7%–48.5%) and 68.8% (95% CI, 57.9%–77.8%) of diseased individuals exhibited a primary or secondary rash, respectively. Distributions of the intrinsic incubation period are shown in figure 1. The mean ± SD values were 6.0 ± 1.4 days (95% CI, 5.6–6.4 days) and 5.7 ± 1.5 days (95% CI, 5.3–6.0 days) in the DENV-4 and DENV-1 studies, respectively, yielding maximum likelihood estimates of the scale...
This study examined several aspects of the natural history of DF based on 2 rigorous historical experiments. These studies are unique in that the experimental details for primary DENV infection were provided and because the etiology was known . These records are the most comprehensive of their kind with regard to the intrinsic and extrinsic incubation and infectious periods. Moreover, it was possible for us to compare signs and symptoms as well as other epidemiologic parameters between serotypes. Three findings were notable. First, this study is the first, to our knowledge, to characterize statistical details of the incubation period of DF. Usually, it is extremely difficult to identify the time of infection for vectorborne diseases. For this reason, the incubation period is conveniently extrapolated from experimental inoculation data, travel histories of cases, or incidence data during the point source outbreak, which offers the time of exposure . Similar data to ours (i.e....