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  1. Amebiasis is a disease caused by a one-celled parasite called Entamoeba histolytica. Who is at risk for amebiasis? Although anyone can have this disease, it is more common in people who live in tropical areas with poor sanitary conditions. In the United States, amebiasis is most common in:

  2. Oct 6, 2022 · Life cycle of Entamoeba histolytica has two-stage: motile trophozoite and non-motile cyst. Trophozoites are found in intestinal lesions, extra-intestinal lesions, and diarrheal stools whereas cyst predominates in non-diarrheal stools.

  3. Dec 2, 2018 · Entamoeba histolytica is an invasive enteric protozoan [ 1, 2, 10 ]. Infection typically begins with the ingestion of mature, quadrinucleated cysts found in fecally contaminated food or water. Excystation occurs in the small intestine with the release of motile trophozoites, which migrate to the large intestine.

  4. Nov 22, 2023 · Intestinal amebiasis is caused by the protozoan Entamoeba histolytica. There are four species of intestinal amebae with identical morphologic characteristics: E. histolytica, E. dispar, E. moshkovskii, and E. bangladeshi [ 1,2 ]. Most symptomatic disease is caused by E. histolytica; E. dispar is generally considered nonpathogenic.

  5. Feb 13, 2017 · Entamoeba histolytica is a unicellular, protozoon parasite of humans. It moves by a jelly-like tongue-like protrusion of the cytoplasm “pseudopodium.” Infection with E. histolytica may be the cause of a variety of symptoms, beginning from no symptoms to severe fulminating intestinal and/or life-threatening extraintestinal disease.

  6. Nov 12, 2020 · E. histolytica is the causative agent of the diarrheal disease known as amebiasis, but it can sometimes penetrate the intestinal wall, enter the circulation, and cause abscesses throughout the body, most commonly in the liver. E. histolytica has 2 main stages during its life cycle: the trophozoite and the cyst stage.

  7. Dec 15, 2022 · Pathogenicity and virulence of Entamoeba histolytica, the agent of amoebiasis. Nancy Guillén. Article: 2158656 | Received 27 Sep 2022, Accepted 11 Dec 2022, Published online: 04 Jan 2023. Cite this article. https://doi.org/10.1080/21505594.2022.2158656. In this article. Full Article. Figures & data. References. Citations. Metrics. Licensing.

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