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  1. Jan 17, 2019 · The onset of a morbilliform eruption (MDE; also known as exanthematous or maculopapular drug eruption) typically occurs within 7 to 10 days after the initiation of the culprit drug. Occasionally a ...

  2. Morbilliform drug eruption is a measles -like rash that develops as a reaction to taking a drug. Any drug can trigger it, but the most common causes are antibiotics. The rash generally goes away after you stop taking the drug, although it may last 1-2 weeks. The rash could be itchy and be accompanied by a low-grade fever.

  3. Morbilliform drug eruption, or reaction, is a skin rash that develops after exposure to certain medications, usually antibiotics like penicillin or cephalosporin. A morbilliform rash is composed of flat pink or red spots that may merge or become raised as the rash spreads. A morbilliform rash often appears on the chest and back first.

  4. Mar 21, 2017 · Drug reactions. Allergic reactions to a drug may be the cause if the maculopapular rash develops four to 12 days after taking a medication. Reactions to medications can take up to seven or eight ...

  5. Jun 16, 2017 · Immune-Related Drug Reactions. Exanthematous Drug Eruptions: Exanthematous drug eruptions, also known as morbilliform or maculopapular drug eruptions, are the most common of all drug-induced reactions. 6 This type of reaction, which is delayed, manifests 1 to 2 weeks after drug initiation but can occur up to 1 week after the drug is stopped; it ...

  6. Drug hypersensitivity syndrome is a specific, severe, unexpected reaction to a medicine, which affects several organ systems at the same time. It typically causes a combination of: High fever; Morbilliform eruption; Haematological abnormalities; Lymphadenopathy; Inflammation of one or more internal organs.

  7. Fixed drug eruption is a delayed type IV hypersensitivity reaction. In the initial phase memory CD8+ T-cells at the dermo- epidermal junction release interferon-gamma when activated by the medication antigen causing epidermal basal layer damage. Recruited T-cells and neutrophils damage melanocytes and keratinocytes.

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